Involvement of interleukin-3 in delayed-type hypersensitivity.

نویسندگان

  • N Mach
  • C S Lantz
  • S J Galli
  • G Reznikoff
  • M Mihm
  • C Small
  • R Granstein
  • S Beissert
  • M Sadelain
  • R C Mulligan
  • G Dranoff
چکیده

The in vivo functions of interleukin-3 (IL-3) were investigated by generating IL-3-deficient mice. Although hematopoiesis was unimpaired in homozygous mutant animals, contact hypersensitivity reactions were compromised. IL-3 was required for efficient priming of hapten-specific contact hypersensitivity responses, but was dispensable for T-cell-dependent sensitization to tumor cells. These findings reveal a critical role for IL-3 in some forms of delayed-type hypersensitivity.

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عنوان ژورنال:
  • Blood

دوره 91 3  شماره 

صفحات  -

تاریخ انتشار 1998